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近年来已明确,沙门氏菌属的内毒素引起小肠上皮细胞分泌增加的作用,取决于内毒素对细胞环核苷酸合成的影响;还有报道提到,在复制的急性肠道感染(包括沙菌感染),在感染者的血液和各种组织内的前列腺素水平大大增高。不少研究证实,前列腺素对抗环核苷酸的合成。故本文作者曾认为,急性沙菌感染导致的机体内环境破坏,与前列腺紊急增有关。众所周知,消炎痛对前列腺素的生物合成具有明显的抑制作用。为了证实消炎痛在沙菌感染时有抑制
In recent years, it has been clarified that the role of endotoxin of Salmonella in increasing the secretion of small intestine epithelial cells depends on the effect of endotoxin on the synthesis of cellular cyclic nucleotides. It has also been reported that in the replication of acute intestinal infections, Infection), the level of prostaglandin in the blood of infected persons and various tissues is greatly increased. Many studies have confirmed that prostaglandin against cyclic nucleotide synthesis. Therefore, the author of this paper once considered that the destruction of the environment caused by acute sand-borne infection is related to the sudden increase of prostate gland. As we all know, indomethacin prostaglandin biosynthesis has a significant inhibitory effect. In order to confirm indomethacin inhibition in the sand infection