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患者男,46岁,患者2001年因眼睑、双下肢水肿,24h尿蛋白定量14.2g,Scr140μmol/L,肾活检为肾小球微小病变,诊断为肾病综合征、肾小球微小病变。经泼尼松60mg/d治疗,尿蛋白转阴、水肿消退,但泼尼松减量至30mg/d时又出现水肿、大量蛋白尿,诊断为肾病综合征复发、激素依赖型。以后继续用泼尼松,加用CTX治疗,尿蛋白转阴,但出现肝功能损害、外周血WBC减少。停用CTX,用肝安、TAD等药物护肝,强力升白片提升WBC
The patient, male, 46 years old, was diagnosed as nephrotic syndrome and glomerular micronest disease because of eyelid and lower extremity edema in 2001, 14.2 g of urinary protein in 24 hours and Scr140 μmol / L in renal biopsy. The prednisone 60mg / d treatment, urine protein negative, edema subsided, but when prednisone reduced to 30mg / d and edema, a large number of proteinuria, diagnosis of nephrotic syndrome relapse, hormone-dependent. After continuing with prednisone, plus CTX treatment, urine protein turned negative, but there is damage to liver function, peripheral blood WBC decreased. Disable CTX, with liver security, TAD and other drugs to protect the liver, enhance the white film or enhance WBC