二氧化碳潴留和低氧血症会引起脑血管扩张,从而造成颅内压(ICP)升高,进而影响脑血流动力学,本文探讨阻塞性睡眠呼吸暂停(OSA)中ICP的变化.作者对6例OSA 患者(呼吸暂停指数>40次/h,暂停时间>20sec),在除外任何神经系疾患(包括颅脑CT 正常)后,进行呼吸监测,并测经皮氧分压(PtcO_2)和二氧化碳分压(PtcCO_2)、动脉压(AP)、中心静脉压(CVP)。经压力传感器测ICP.此外还测了脑电图和颏肌电图。4例患者清醒时ICP 超过正常(>15mmHg),晨间又比傍晚略高.睡眠中平均ICP 和AP 均较清醒时高。在非快速 Carbon dioxide retention and hypoxemia cause cerebral vasodilatation, resulting in increased intracranial pressure (ICP), which in turn affects cerebral hemodynamics, and this article discusses changes in ICP in obstructive sleep apnea (OSA) Patients with OSA (apnea index> 40 beats / h, pause time> 20sec) and respiratory monitoring were performed after exclusion of any neurological disorders (including normal brain CT), and the percutaneous oxygen pressure (PtcO_2) and carbon dioxide Partial pressure (PtcCO_2), arterial pressure (AP), central venous pressure (CVP). Measured by pressure sensor ICP. In addition, the electroencephalogram and chin EMG also measured. In 4 patients, the ICP was more than normal (> 15mmHg) when awake, and slightly higher in the morning than in the evening. Mean ICP and AP during sleep were higher than those during awake. Not fast