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目的本研究通过随机抽样调查18~22岁肥胖人群骨密度、上气道体积变化、以及比较肥胖和正常体重人群安氏Ⅱ类错畸形牙特征差异,旨在分析肥胖因素对人群牙特征的可能影响。方法对1024例18岁~22岁人群进行体重指数(BMI)的检查及安氏Ⅱ类错畸形筛查,BMI值≥23且合并安氏Ⅱ类错畸形人群100例构成肥胖组,在剩余BMI值<23的人群中,抽取100例伴有安氏Ⅱ类错畸形人群作为配对正常组。分别对两组人群进行骨密度的检查分析,以评估两组人群骨量的发育情况;分别对两组人群进行X线头颅定位侧位片的拍摄,测量上气道体积;制取口腔模型并作模型分析,测量牙弓拥挤度、牙弓宽度、SPEE曲度等指标。结果 (1)肥胖组人群的T值(t=4.18,P<0.05)和Z值(t=3.86,P<0.05)均明显低于正常组;(2)肥胖组的PNS-R(t=12.88,P<0.05)、PNS-UPW(t=23.91,P<0.05)、SPP-SPPW(t=37.54,P<0.05)、U-MPW(t=66.16,P<0.05)、TB-TPPW(t=34.91,P<0.05)和V-LPW(t=10.86,P<0.05)均明显低于正常组人群;(3)肥胖组与对照组的spee曲度不存在统计学差异(P>0.05);(4)肥胖组的上颌后牙弓宽度明显大于正常组(Z=-2.196,P=0.027<0.05),下颌中牙弓宽度肥胖组明显小于正常组(Z=-2.164,P=0.030<0.05),其他部位不存在统计学差异(P>0.05);(5)肥胖组下颌的牙列拥挤度明显大于正常组(Z=-2.765,P=0.018<0.05,上颌牙列拥挤度不存在统计学差异(P>0.05)。结论肥胖能引起人群骨密度降低,上气道狭窄。骨骼发育与气道体积减小都可能影响人群牙发育。进一步对肥胖组人群的牙特征分析发现,该类型人群下牙弓更加拥挤,牙弓宽度减小,这一变化与肥胖人群骨量发育不足,气道体积减小等改变相符合,提示肥胖与某些类型错畸形的发生发展可能存在影响关系。但是本次调查得到正常组与肥胖组安氏Ⅱ类错畸形发病率并无统计学差异,尚不能证实这一推测是否正确,有待进一步的探究。
Objective To investigate the changes of bone mineral density (BMD) and upper airway volume (LVEF) in obese people aged 18-22 years and the differences in malocclusion of Class Ⅱ malocclusion between obese and normal weight population in a random sample to analyze the effect of obesity on population characteristics May affect. Methods A total of 1024 BMIs and Class Ⅱ malocclusion screening were performed in 1024 adults aged 18 to 22 years with a BMI of ≥23 and 100 patients with Class Ⅱ malocclusion. Of the remaining BMI values <23, 100 patients with Class Ⅱ malocclusion were selected as matched normal subjects. The bone mineral density of the two groups were examined and analyzed to evaluate the development of bone mass in the two groups. The X-ray skull positioning lateral radiographs were taken to measure the upper airway volume of the two groups. For model analysis, dental arch crowding, dental arch width, SPEE curvature and other indicators. Results (1) The T value (t = 4.18, P <0.05) and Z value (t = 3.86, P <0.05) in obese group were significantly lower than those in normal group; 12.88, P <0.05); PNS-UPW (t = 23.91, P <0.05); SPP-SPPW t = 34.91, P <0.05) and V-LPW (t = 10.86, P <0.05) were significantly lower than those in the normal group; (3) There was no significant difference in spee curvature between the obesity group and the control group ); (4) The maxillary posterior arch width in obese group was significantly larger than that in normal group (Z = -2.196, P = 0.027 <0.05) (P <0.05). There was no statistical difference in other sites (P> 0.05). (5) The dentition crowding of mandibular dentition in obesity group was significantly greater than that in normal group (Z = -2.765, P = 0.018 < (P> 0.05) .Conclusion Obese can cause population density decrease, upper airway stenosis, skeletal development and airway volume decrease may affect the population development of teeth.Finally, This type of population under the more crowded dental arch, dental arch width decreases, this change and obesity in bone mass development, airway volume reduction and other changes Disguised coincidence, suggesting that obesity and some types of malformations deformity may have an impact on the occurrence and development.However, this survey was normal and obese group of Class Ⅱ malformations Malformation incidence was not significantly different, yet can not confirm this Speculation is correct, to be further explored.