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用去除心交感神经、迷走神经和静注心得安、酚妥拉明或小剂量异丙基肾上腺素等方法,观察植物神经在刺激下丘脑背内侧核(DMH)诱发“缺血样”心电改变中的作用。结果:1.切断颈两侧迷走神经,对“缺血样”心电改变无明显影响。2.去除双侧星状神经节及胸2～4椎旁交感神经节,可减轻已形成的“缺血样”心电改变并阻止再刺DMH时ST段的进一步降低,3.静注心得安、酚妥拉明不能阻断“缺血样”,心电改变。4.静注异丙基肾上腺素使部分动物暂时消除“缺血样”心电改变。5.用平均动脉压与心率的乘积代表心肌氧耗量,表明“缺血样”心电改变的形成与氧耗增加无明显关系。
With the removal of cardiac sympathetic nerve, vagal and intravenous propranolol, phentolamine or low-dose isoproterenol and other methods to observe the autonomic nerve stimulation in the dorsal medial hypothalamus (DMH) induced “ischemic” ECG changes In the role. Results: 1. Cut off the vagus nerve on both sides of the neck, the “ischemic” ECG changes had no significant effect. 2. Removal of bilateral stellate ganglion and chest paraspinal sympathetic ganglia 2 to 4, can reduce the formation of “ischemic” ECG changes and prevent further stabbing DMH ST segment further reduction, 3. intravenous experience Ann, phentolamine can not block the “ischemic” ECG changes. 4. Intravenous isoproterenol so that some animals temporarily eliminate the “ischemic” ECG changes. 5. The mean arterial pressure and heart rate product of myocardial oxygen consumption, indicating that “ischemic” ECG changes in the formation of oxygen consumption increased with no significant relationship.