论文部分内容阅读
本研究建立犬心肌缺血再灌注模型,用二维超声心动图(2DE)测量的节段性室壁增厚率(△T)作为反映局部心肌收缩功能的指标,以M型超声心动图测量的舒张末期室壁变薄一半时的平均速率(RHEDT)作为反映局部心肌舒张功能的指标,定量评价了阻断犬冠状动脉左前降支(LAD)15min,再灌注180min时的心功能变化。结果表明:眩晕心肌局部收缩功能和舒张功能均受到抑制,功能的恢复为一渐进过程,两者的变化呈平行发展的趋势。
Myocardial ischemia-reperfusion model was established in this study. The segmental ventricular wall thickening (△ T) measured by two-dimensional echocardiography (2DE) was used as an index to reflect the local myocardial contractile function. M-mode echocardiography (RHEDT) as the index of local myocardial diastolic function, quantitatively evaluated the changes of cardiac function after occluding canine left anterior descending artery (LAD) for 15 minutes and reperfused for 180 minutes. The results showed that both local systolic and diastolic functions of vertigo myocardium were inhibited and the recovery of function was a gradual process, and the changes of both were parallel.