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微循环学说认为,各种休克动因作用于机体,都可以导致微循环灌流不足,组织缺血缺氧,继而微循环淤滞,出现弥漫性血管内凝血(DIC),以致细胞代谢严重障碍,器官功能受损,使休克的病理过程趋向不可逆发展。那么休克时微循环紊乱的基础是什么?近年来的研究表明,休克时神经—体液因素,尤其是体液因素,种类繁多,效应广泛,而且彼此关联,互相影响,是造成微循环障碍的主要原因。有人曾设想,儿茶酚胺(CA)、加压素、血管紧张素Ⅱ(AⅡ)、5-羟色胺(5—HT)、内源性阿片样肽(EOP)、组胺、缓激肽(BK)、前列腺素(PG)、心肌
Microcirculation theory that a variety of shock motivation in the body, can lead to inadequate microcirculation, tissue ischemia and hypoxia, followed by microcirculation, diffuse intravascular coagulation (DIC), resulting in serious cell metabolism disorders, organ function Damaged, the pathological process of shock tends to irreversible development. So the basis of microcirculation disturbance in shock is what? Recent studies have shown that neurological - humoral factors, especially the body fluid factors, a wide range of effects, a wide range of effects, and associated with each other and affect each other, is the main cause of microcirculation disorders . It has been proposed that catecholamine (CA), vasopressin, angiotensin II (AII), serotonin (5-HT), endogenous opioid peptide (EOP), histamine, bradykinin (BK) Prostaglandin (PG), myocardium