肾小管性酸中毒(Renal tubulal acido-sis)系肾性酸中毒的一种罕见类型,近年颇引起医学界的重视,成年的病例在国内亦曾有报导。兹将我院所遇到的以血钾过低所致肌麻痺为突出表现的1例报告如下:患者郜××,住院号45626,女,29岁,工人,因半年来发作性周身瘫痪及骨骼疼痛加甚,于1963年6月6日入院。患者于1958年冬天,自觉四肢乏力,不能胜任日常劳动;半个月后某晚,突然全身瘫痪,当时神志清,感觉无异常。经服氯化钾治疗,三天后即能行走。1959年2月又有同样发作,服氯化钾而缓解。1960年冬天,第三次瘫痪发生,并伴有多尿,口渴,多饮等症状,又服氯化钾数天后瘫痪缓解,但多尿仍有。1961年9月下旬第三次妊娠分娩后出现走路时两足跟部疼痛,1962年初疼痛发展到两膝及腰部,活动时疼痛加剧,但与天气无关,亦非游走性。同年冬天,全身躯干及四肢骨骼、关节均有明显疼痛,稍微活动即引起剧烈疼痛,腿不能跨大步,手不能提物。1963年1月17日以后因全身骨痛加剧及发作性周身瘫痪而被迫 Renal tubulal acidosis (Renal tubulal acido-sis) is a rare type of renal acidosis, caused considerable medical attention in recent years, adult cases have also been reported in the country. I will encounter in our hospital to hypokalemia due to muscle paralysis as a prominent manifestation of a case report is as follows: Patient × ×, hospital 45626, female, 29 years old, workers, due to the onset of six weeks of systemic paralysis and Bone pain plus, in June 6, 1963 admitted. Patients in the winter of 1958, conscious limb weakness, can not be competent for daily work; one night after half a month, suddenly paralyzed, then conscious, feeling normal. After serving potassium chloride treatment, can walk after three days. 1959 February the same attack, serving potassium chloride and ease. The winter of 1960, the third paralysis occurred, and accompanied by symptoms of polyuria, thirst, polydipsia and then paralyzed a few days after serving potassium chloride, but there is still polyuria. In the second trimester of September 1961, there was pain in the two feet and heel when walking after delivery. In early 1962, the pain developed to both knees and waist, and the pain was aggravated during the activity. However, it had nothing to do with the weather or migration. The same winter, the whole body and limbs bones, joints were obvious pain, a little activity that caused severe pain, legs can not be stride, hands can not mention material. After January 17, 1963, the whole body was aggravated by bone pain and paroxysmal episodes