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本文探讨用线粒体的分子热机工作原理研究运动疲劳 ,从呼吸链电子漏影响线粒体合成ATP效率的角度对运动疲劳发生的分子机制做出解释 ,并分析其影响因素 ;在基因水平上分析运动训练对运动员机体的影响 ,指出运动应激产生的活性氧对运动员机体造成的基因损伤、修复和复制更新机制及活性氧作为信号分子通过氧化还原修饰传递相关刺激信号激活转录因子诱导基因表达是运动员通过运动训练提高体能和改变身体形态的分子机制 ,并提出运动训练的“弹弓”假说 ,为科学化训练提供一个思路 ;从基因表达调控和线粒体蛋白质组学的角度分析运动员科学选材的依据 ,提出建立运动员科学选材的理论基础。
In this paper, we study the mechanism of mitochondrial thermokinetic study on exercise fatigue, explain the molecular mechanism of exercise fatigue from the perspective of the effect of respiratory chain electron leakage on mitochondrial ATP synthesis, and analyze the influencing factors. At the gene level, Athletes, pointed out that exercise stress caused by reactive oxygen species of the body caused by athletes damage, repair and replication mechanisms and reactive oxygen species as signaling molecules through the redox modification of the relevant stimulus signal activation of transcription factors induced gene expression is the movement of athletes through exercise Training to improve physical fitness and change the physical form of the molecular mechanism, and proposed sports training “slingshot” hypothesis for scientific training to provide a train of thought; from gene expression regulation and mitochondrial proteomics point of view of the scientific selection of athletes, put forward to establish athletes The theoretical basis of scientific material selection.