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在以往的许多生理学实验中，尿生成的影响因素实验常因无尿而使实验失败，而且持续严重的失血休克可导致肾功能障碍，尿量减少，甚至无尿。临床上在休克后给予补充生理盐水等各种措施可恢复血压，维持各种生命活动。Ｌ－精氨酸是生成ＮＯ的底物，ＮＯ是一种有许多生理、病理作用的气体物质，ＮＯ可扩张血管，改善肾循环，从而对尿的生成有影响及对肾功能有保护作用。本实验观察了Ｌ－精氨酸对家兔尿生成的影响，观察了腹腔注射Ｌ－精氨酸组与对照组在生理条件下及失血性休克后的尿量，做ｔ检验，结果显示有差异
In many physiological experiments in the past, experiments on the factors influencing the formation of urine often failed due to anuria, and continued severe hemorrhagic shock led to renal dysfunction, decreased urine output and even no urine. Clinically after the shock given saline and other measures to restore blood pressure, to maintain a variety of life activities. L-arginine is the substrate for the production of NO. NO is a gaseous substance that has many physiological and pathological effects. NO can dilate blood vessels and improve renal circulation, thus affecting the formation of urine and protecting renal function. This experiment observed the L-arginine on the urine of rabbits, observed the intraperitoneal injection of L-arginine group and the control group under physiological conditions and post-hemorrhagic shock urine output t-test showed that there difference