论文部分内容阅读
前文报告因中枢经α-肾上腺素受体激动而产生的降压作用有可能是通过γ-氨基丁酸(GABA)能抑制性神经元而实现。鉴于β肾上腺素受体拮抗剂心得安的抗高血压效应已被临床肯定,而且近年又报告心得安引起的降压作用伴有内脏神经放电减少;在几个脑区
The previous report that the central hypothalamic effect due to α-adrenergic receptor activation may be via γ-aminobutyric acid (GABA) inhibitory neurons. In view of β-adrenergic receptor antagonist propranolol antihypertensive effect has been clinically affirmed, but also reported in recent years, propranolol caused by the hypotensive effect associated with reduced visceral nerve discharge; in several brain regions