图像视诱发电位(简称VEP)已经表明对脱髓鞘性病变、局部缺血和前视路受压迫引起的视神经损害很敏感。据报道青光眼也可使VEP幅度降低及潜伏期延长,VEP潜伏期延长与杯陷增大和视野缺损有关。高眼压者的VEP正常,除非使用激发技术或偏心观察。在那些没有用激发技术的研究中,VEP潜伏期的异常延长,则不清楚是否部分地由于缩瞳、高龄或视力减退所干扰。这三个因素均能使VEP潜伏期延长。曾有研究对此三因素均经精细控制,报道了青光眼患者与正常对照者VEP潜伏期有小的组差。 Visual evoked potentials (VEPs) have been shown to be sensitive to demyelinating lesions, ischemia, and optic nerve damage caused by compression of the anterior optic path. It is reported that glaucoma can also reduce the VEP amplitude and latency, prolonged VEP latency and increased cup trauma and visual field defects. High intraocular pressure of the VEP normal, unless the use of excitation techniques or eccentric observation. In those studies that did not use excitation techniques, the abnormal prolongation of the VEP latency was not clear if it was partly due to miosis, age or vision loss. All three of these factors prolong the latency to VEP. There have been studies of these three factors are finely controlled, reported in patients with glaucoma and normal controls were a small difference in latency of VEP.