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患婴幼儿肺炎时由于缺氧、酸中毒及肺分泌血管活性物质增多,引起肺动脉及小动脉痉挛,产生肺动脉高压.以上病理生理改变得不到及时纠正则可产生心力衰竭.近年来由于对心衰基础研究的不断深入,治疗对策有很大进展,尤其是血管扩张剂和非糖甙类正性肌力药物及山莨菪类等血管活性药物临床应用,改变了传统肺炎心衰的治疗.既往常规治疗主要应用
Suffering from infant pneumonia due to hypoxia, acidosis and pulmonary secretions of vasoactive substances increased, causing pulmonary and arteriolar spasm, resulting in pulmonary hypertension. The above pathophysiological changes can not be corrected in time can produce heart failure. In recent years due to the heart The deepening of the research on the basis of decay, great progress has been made in the treatment strategies, especially the clinical application of vasodilators such as vasodilators and non-glycoside inosotics and anisodamine, which have changed the treatment of traditional pneumonia with heart failure. The main application of conventional treatment