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目的分析获得性长Q-T间期综合征的临床特点。方法对90例获得性长Q-T间期综合征病例的临床资料进行回顾性分析。结果 90例中女60例,男30例,发作尖端扭转型室性心动过速1～7/人,平均3.2次,共计29次的尖端扭转型室性心动过速发作中,6次为电击除颤转复(20.7%),其余自行转复;8例应用异丙肾上腺素静脉滴注使心率维持在90～110/min,随后对其中3例植入临时起博器,给予90～110次/min的频率持续右心室心尖部起博,同时去除诱发因素,如纠正低钾血症,补镁,停用胺碘酮及其他引起Q-T间期延长的药物。结论获得性长Q-T间期综合征为心源性晕厥愿因之一,常诱发尖端扭转型室性心动过速,补钾、镁为重要错施。
Objective To analyze the clinical features of acquired long Q-T syndrome. Methods The clinical data of 90 patients with acquired long Q-T syndrome were retrospectively analyzed. Results 90 cases of female 60 cases, 30 males, onset of torsades de pointes ventricular tachycardia 1 ~ 7 / person, an average of 3.2 times, a total of 29 times in the torsades de pointes tachycardia attack, 6 times as electric shock Defibrillation (20.7%), the rest of the self-transfer; 8 cases of intravenous infusion of isoproterenol to maintain heart rate 90 ~ 110 / min, followed by 3 cases were implanted in a temporary pacemaker, given 90 ~ 110 The frequency of min / min continued right apex apical pacing, while removing predisposing factors, such as the correction of hypokalemia, magnesium supplements, the withdrawal of amiodarone and other drugs that cause QT prolongation. Conclusion Acquired long-term Q-T syndrome is one of the causes of cardiogenic syncope. It often induces torsades de pointes and potassium and magnesium are important mistakes.