70年代人们发现嗜酸细胞(EC)能酶解哮喘速发反应(EAR)中的一些炎性介质,故认为哮喘病人支气管-肺组织中EC 的增多是机体正常的一种免疫调节机制,具有保护作用.近年来,炎症与哮喘的关系日益受到重视.气道的炎症可以提供某些条件或其本身就是引起气道高反应性的机制。哮喘病人气道的主要组织病理改变包括:支气管粘膜的水肿:纤毛上皮损伤、脱落和EC 浸润。Reed 把哮喘称为 70s people found that eosinophils (EC) can be enzymatic reaction of asthma rapid response (EAR) in some of the inflammatory mediators, so that bronchial-lung EC in asthma patients is an increase of the body’s normal immune regulatory mechanism, with Protection In recent years, the relationship between inflammation and asthma has been paid more and more attention.The airway inflammation can provide certain conditions or is itself a mechanism that causes airway hyperresponsiveness. The main histopathological changes in asthmatic airways include: bronchial mucosal edema: ciliated epithelial damage, shedding and EC infiltration. Reed called asthma