经典理论认为,胸腔内液体来自胸膜毛细血管;漏出液系毛细血管内静水压与渗透压之间不平衡所致,此种液体蛋白质含量低;渗出液系各种原因引起的胸膜毛细血管通透性增加,使液体进入胸腔,此种液体蛋白质含量高。目前则认为,这一理论应予修正。 关于静水压性肺水肿的最新研究表明,漏出液来自肺间质腔隙。给绵羊超负荷液体造成肺水肿后,可见液体自脏层胸膜进入胸腔。其内的蛋白质含量与肺淋巴液及间质液中的蛋白质含量一样,液体量约为总肺水肿液体的25％。用其他方 The classic theory that the fluid in the chest cavity from the pleural capillary; leakage of liquid capillary within the hydrostatic pressure and osmotic pressure caused by the imbalance between the liquid protein content is low; exudate of various causes of pleural capillaries Permeability increases, the liquid into the chest, the liquid protein content is high. At present, we think that this theory should be amended. Recent studies on hydrostatic pulmonary edema have shown that the leakage is from interstitial lung space. To the sheep caused by fluid overload caused by pulmonary edema, visible liquid from the visceral pleura into the chest. Its protein content and lung lymph and interstitial fluid protein content, the amount of fluid is about 25% of the total pulmonary edema fluid. Use other party