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在实验生态条件下 ,浓度范围 0 .1～ 5 0 μg/L的苯并 (a)芘和芘的短期暴露 (7d) ,5 0 μg/L浓度组造成梭鱼肝脏SOD活性先抑制后诱导的效应 ;5 μg/L浓度在 7d的暴露中 ,SOD活性未出现诱导而是抑制 ;同样在 5 0μg/L浓度下 ,苯并 (a)芘暴露 4d后SOD活性出现诱导 ,而芘在暴露 7d后才出现诱导 ,这间接反映了苯并 (a)芘和芘的毒性大小。这些结果说明梭鱼肝脏SOD活性与苯并 (a)芘和芘暴露有一定的相关性 ,可以作为海洋环境多环芳烃污染监测的一种生物标志物。
Under the experimental ecological conditions, the short-term exposure (7d) of benzo (a) pyrene and pyrene in the concentration range of 0.1-150 μg / L and the concentration of 50 μg / L led to the inhibition of SOD activity in the liver (P <0.05). Under the concentration of 5 μg / L, the activity of SOD was not induced but inhibited at the concentration of 5 μg / L for 7 d. Under the same concentration of 5 μg / L, the activity of SOD was induced after exposure to benzo (a) Induction occurred after 7 days, which indirectly reflects the toxicity of benzo (a) pyrene and pyrene. These results indicate that there is a certain correlation between the activity of SOD and the exposure to benzo (a) pyrene and pyrene in the liver of barracuda so that it can be used as a biomarker to monitor the PAHs pollution in marine environment.