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美国心脏协会下属杂志《循环》报道,英国布里斯托尔大学等科研机构的研究人员发现,心脏病发作时,心脏神经系统会产生疼痛反应,并会释放一种名为“P物质”的分子,它会激发身体的自我修复能力,将骨髓中的干细胞移动到心脏缺氧的受损部位。接着,干细胞形成新的血管,为心脏提供新鲜的血液。布里斯托尔大学的保罗·马代杜教授表示,使用止痛药抑制疼痛后,上述修复过程就不能正常进行,从长期来说,不利于患者康复。此前的研究也表明,心脏病
Researchers at a research institute such as the University of Bristol in the United Kingdom found that the heart nervous system produces a painful response to a heart attack and releases a substance called “substance P” It stimulates the body’s ability to repair itself, moving stem cells in the bone marrow to damaged parts of the heart that lack oxygen. The stem cells then form new blood vessels that provide the heart with fresh blood. Professor Paul Maddoud of the University of Bristol said that the use of analgesics to suppress pain, the repair process can not be normal, in the long run, is not conducive to patient rehabilitation. Earlier studies have also shown that heart disease