急性心肌梗塞(AMI)传统的治疗重点在于防治致死性心律失常和心泵衰竭等并发症,而纠正心肌氧供需失衡,挽救濒死心肌,缩小梗塞面积。改善存活者远期预后和提高生活能力,则远未真正解决。近年来,既重视降低心肌氧需以保护缺血心肌,更强调唯有恢复冠状动脉(冠脉)灌注,增加心肌氧供,始可能最大限度地挽救濒死心肌、缩小梗塞面积、改善整体和局部的左室功能。为此,AMI治疗立足于“一早二远”。一早:早期诊断后,力争在心肌出现不可逆性坏死之前,尽早重建冠脉血流,恢复缺血区再灌注:二远,改善AMI存活者 The traditional treatment of acute myocardial infarction (AMI) focuses on the prevention and treatment of complications such as fatal arrhythmia and heart failure, and to correct myocardial oxygen supply and demand imbalance to save the dying myocardium and reduce infarct size. To improve the long-term survival of survivors and improve their ability to live, is far from real solution. In recent years, both the importance of reducing myocardial oxygen to protect the ischemic myocardium, but more emphasis on the restoration of coronary artery (coronary artery) perfusion and increase myocardial oxygen supply may be to maximize the rescue of dying myocardium, infarct size and improve overall Local left ventricular function. To this end, AMI treatment based on “a long way to go.” Early morning: After early diagnosis, and strive to myocardial irreversible necrosis before the early reconstruction of coronary blood flow, reperfusion ischemia area to resume: two far, to improve AMI survivors