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格林巴利综合征(Guillain-Barre syndrome,GBS)是一种自身免疫性周围神经疾病。促炎性因子与抑炎性因子通过改变Th1/Th2型细胞之间的平衡,主要是增强Th1型细胞的作用,在疾病的发生中起着重要的作用。它们的平衡影响着疾病的发生、发展和转归。
Guillain-Barre syndrome (GBS) is an autoimmune peripheral neuropathy. Pro-inflammatory and anti-inflammatory cytokines play an important role in the pathogenesis of Th1 cells by changing the balance between Th1 / Th2 cells. Their balance affects the occurrence, development and outcome of the disease.