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炎症反应是动脉粥样硬化(atherosclerosis,AS)的重要发病机制,并与AS进程中斑块不稳定、破裂和继发血栓形成等急性血管事件的发生密切相关[1]。众多证据表明,血管紧张素(angiotensin,Ang)Ⅱ通过激活氧化应激反应生成反应性活性氧族
Inflammatory reaction is an important pathogenesis of atherosclerosis (AS) and is closely related to the occurrence of acute vascular events such as plaque instability, rupture and secondary thrombosis in AS process [1]. Numerous evidences indicate that angiotensin (Ang) Ⅱ generates reactive reactive oxygen species by activating oxidative stress