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自1800年代以来对于青光眼视野缺损和视盘凹陷形成的原因一直存在着争论。一派认为眼压升高压缩筛板前和筛板部纤维而阻断神经轴索是青光眼神经损害的基本原因;另一派不否认眼压升高在部分病人中的重要性,但指出有些病人并不存在眼压升高,因而提出血流供应不足引起的营养障碍导致视神经改变的理论。一、青光眼视野缺损的血管学说近年来血管学说因得到许多杰出学者的支持而居统治地位。该说不仅认为青光眼视野缺损和凹陷是由于视神经乳头血供减少所致,而且还认为全身性血管疾患也是该过程的重要因素,特别对于低眼压性青光眼。Duke-Elder(1941)就曾指出:“必须强调,原发性青光眼不是眼的局部
There have been controversies about the causes of glaucomatous visual field defects and disc degeneration since the 1800’s. One group believes that the intraocular pressure increased compression of the frit and the frit section of the fiber optic nerve block axon injury is the basic reason for glaucoma nerve damage; the other party does not deny the increased intraocular pressure in some patients the importance of, but pointed out that some patients do not There is a rise in intraocular pressure, thus suggesting that the nutritional deficits caused by inadequate blood flow lead to a theory of optic nerve changes. First, the glaucoma vascular vision defects that vascular theory in recent years due to the support of many outstanding scholars and dominance. This is said to be not only due to glaucomatous visual field defects and depressions caused by decreased optic nerve head blood supply, but also that systemic vascular disease is also an important factor in this process, especially for low pressure glaucoma. Duke-Elder (1941) once pointed out: "It must be emphasized that primary glaucoma is not part of the eye