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最近作者曾报告动脉瘤致蛛网膜下腔出血病人血浆粘度与脑缺血的关系.静滴低分子右旋糖酐(LMWD)250ml每日两次,平均9天,观察到血浆粘度从治疗前的1.38±0.09mPa/秒,至第七天上升到最高值1.66±0.09mPa/秒,以后又渐降低至第12天时为1.56±0.13mPa/秒.已发现有局灶性神经功能缺失的患者其血浆粘度的升高比无局灶性神经功能缺失患者明显.据此,作者断定:高血浆粘度在脑缺血发生上可能起促进作用.但是作者对血浆粘度增高的可能原因未作评价.Kroemer等用LMWD治疗缺血性脑卒中,也观察到治疗10天期间血浆粘度有类似的升高(开始500m
The authors recently reported the relationship between plasma viscosity and cerebral ischemia in patients with aneurysm-induced subarachnoid hemorrhage: intravenous infusion of low molecular weight dextran (LMWD) 250ml twice daily for an average of 9 days and a change in plasma viscosity from 1.38 ± 0.09 mPa / s, rose to a maximum of 1.66 ± 0.09 mPa / s on the seventh day, then gradually decreased to 1.56 ± 0.13 mPa / s on the 12th day, and patients with focal neurological deficits have been found to have plasma viscosity The authors conclude that high plasma viscosity may play a role in promoting the development of cerebral ischemia, but the authors do not evaluate the possible causes of increased plasma viscosity.Kroemer et al LMWD treatment of ischemic stroke also observed similar increases in plasma viscosity during the 10-day treatment (starting at 500 m