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肾上腺素（Ｅ）和去甲肾上腺素（ＮＥ）分别在高于１ｎｍｏｌ／Ｌ和１０ｎｍｏｌ／Ｌ浓度时增强淋巴细胞的增值反应，而皮质酮（ＣＳ）则呈现双相效应，在低于０．１ｎｍｏｌ／Ｌ时促进，高于１０ｎｍｏｌ／Ｌ时抑制此反应．ＣＳ分别与Ｅ或ＮＥ或二者以各自的无效浓度（０．１ｎｍｏｌ／Ｌ）合用时使淋巴细胞的增殖反应显著增强，而且此种增强效应在７５ｍＧｙＸ射线全身照射后进一步上调．乙酸豆蔻波酯（ＰＭＡ）和Ａ２３１８７亦有促进淋巴细胞增殖反应的作用．作者发现，７５ｍＧｙＸ射线全身照射后，ＰＭＡ和Ａ２３１８７所引起的淋巴细胞增殖反应增强进一步上调．这些新实验资料对分析低剂量辐射增强免疫的机制可能具有重要意义．
Epinephrine (E) and norepinephrine (NE) increased lymphocyte response at concentrations higher than 1 nmol / L and 10 nmol / L, respectively, whereas corticosterone (CS) exhibited biphasic effects below 0. 1nmol / L when the promotion, higher than 10nmol / L inhibition of this reaction. CS significantly increased lymphocyte proliferation response when combined with E or NE, respectively, at their respective concentrations (0.1 nmol / L), and this enhancement was further up-regulated after 75 mGy X-ray irradiation. Cardanol acetate (PMA) and A23187 also promote lymphocyte proliferation response. The authors found that PMA and A23187 induced a further upregulation of lymphocyte proliferation response following 75 mGy X-ray whole body irradiation. These new experimental data may have important implications for the analysis of the mechanisms by which low-dose radiation enhances immunity.