长期以来已认识到,偏头痛发作时伴发短暂性神经症状缺损,如麻木、虚弱或失语等。偏头痛的症状持续24小时以上,有可能脑组织梗塞。青年人的短暂性缺血发作(TIAs)可能为良性血管性疾病所致。Preatfield 提出,偏头痛先兆与 TIAs 二者可能伴随血小板凝聚反应后微栓子形成。典型偏头痛发作与TIAs 的区别在于患者对“扩散性皮质抑制”的敏感性。可以认为偏头痛是神经血管反应。在国际头痛学 It has long been recognized that migraine attacks accompany transient neurological deficits such as numbness, weakness or aphasia. Migraine symptoms last more than 24 hours, there may be cerebral infarction. Transient ischemic attacks (TIAs) in young people may be caused by benign vascular disease. Preatfield proposed that both migraine aura and TIAs may be accompanied by micro-emboli formation following platelet aggregation. The difference between a typical migraine attack and TIAs is the patient’s sensitivity to “diffuse cortical inhibition.” Migraine can be considered a neurovascular response. Headache in the world