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有关心肌缺血再灌流损伤的发生机制多倾间于自由基学说。已有资料证实,外源性自由基发生体系FeSO_4/抗坏血酸可损伤心肌线粒体,但该体系在心肌缺血再灌流损伤中所起的作用却很少报道。本实验
The mechanism of myocardial ischemia-reperfusion injury lean more in the free radical theory. It has been confirmed that exogenous free radical generation system FeSO 4 / ascorbic acid can damage myocardial mitochondria, but the role of this system in myocardial ischemia-reperfusion injury has been rarely reported. this experiment