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小鼠每天吸 O_8(0.9ppm)12小时,每晨接受气功外气半小时,连续12天后,测肺、肝组织的过氧化脂产物 MDA 含量和抗氧化酶 GSH-Px 活性,检查肺、肝及免疫器官胸腺和脾脏的病变,以观察气功外气抗 O_3损伤的作用。结果:O_3吸入不仅对肺,也对其它脏器造成损伤,O_3组小鼠肺、肝 MDA 增多,GSH-Px 活性降低,提示小鼠处于氧化性应激状态。气功组小鼠肺。肝内 MDA 少于、GSH-Px 活性高于 O_3组,氧化性应激状态明显改善,肺、肝炎症亦轻。O_3组免疫器官缩小、减重,胸腺和脾脏 T 淋巴细胞群萎缩明显,气功组变化不明显,提示气功外气有保护小鼠抗 O_3损伤的生物学效应。
The mice received O_8 (0.9ppm) daily for 12 hours, received qigong outside the air for half an hour each morning, and after 12 consecutive days, measured the MDA content of lipid peroxidation product of the lungs and liver tissues and the activity of antioxidant enzyme GSH-Px. The lungs and liver were examined. And immune organ thymus and spleen lesions, to observe the effect of Qigong external gas against O_3 injury. RESULTS: O 3 inhalation not only caused damage to the lungs but also to other organs. The MDA in the lungs and liver of the O 3 mice increased and the GSH-Px activity decreased, suggesting that the mice are in oxidative stress. Qigong group mice lungs. With less intrahepatic MDA and higher GSH-Px activity than the O_3 group, oxidative stress was significantly improved, and lung and liver inflammation were also mild. The immune organs in the O3 group were reduced and weight loss. Thymus and spleen T lymphocyte populations were significantly atrophied. The Qigong group did not change significantly, suggesting that the Qigong external gas has the biological effects of protecting mice against O3 damage.