心外膜冠状动脉痉挛可引起短暂的心肌缺血,甚至心肌梗塞及心室纤颤。心外膜冠脉舒缩张力受许多生理机制的影响,这些机制包括:植物神经系统、前列腺素、花生四烯酸的其它代谢产物、血小板因子、血流以及内皮调节的反应。在活体内,心肌缺血可继而增加神经体液刺激物,从而影响并非直接遭受缺血的血管。至令还没有报导心肌缺血对非直接遭受缺血的心外膜冠状动脉的影 Epicardial coronary spasm can cause transient myocardial ischemia, and even myocardial infarction and ventricular fibrillation. Epicardial coronary systolic tensions are impacted by many physiological mechanisms, including the autonomic nervous system, prostaglandins, other metabolites of arachidonic acid, platelet factors, blood flow, and endothelial modulation. In vivo, myocardial ischemia can in turn increase neurohumoral stimuli, affecting blood vessels that are not directly subjected to ischemia. It has not yet been reported that myocardial ischemia has not been directly affected by ischemic epicardial coronary artery shadow