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正常情况下前庭系统和视系统的合作使视靶成像于中央凹。病理情况时反射性眼动和视系统可能互相冲突。视-前庭间缺乏联系影响暗处和亮处之间前庭性眼震强度的减弱,其程度随中枢神经系统病变的部位而改变。通常以视固定指数的百分率表示。视抑制失败提示颅后窝病变,可能是苔状纤维至绒球的输入有缺陷,造成前庭动眼反射对视刺激不敏感。本文测定10例小脑疾患视觉抑制温度性眼震的能力并与跟踪增益和叠加扫视数联系起来,分析前庭增益——椅速除眼速的商,进而描述一种眼-动模型以助理解这些问题。结果:(1)温度性眼震,10例中6例视抑制减弱,有的得负值。(2)平滑跟踪,10例中4例增益明显减低,叠加扫视数增加。(3)前庭性眼震,6例前庭增益增高但与平滑跟踪增益无关。
Under normal circumstances the vestibular system and visual system cooperation so that the visualization of the fovea. Reflective ophthalmic and visual systems may be in conflict with each other under pathological conditions. The lack of visual-anastomosis influences the reduction of vestibular nystagmus between dark and bright spots, to a degree that varies with the site of central nervous system disease. It is usually expressed as a percentage of the fixed index. Suppression of failure as prompted the posterior fossa lesions may be moss fiber to the pompon importation of defects, resulting in the Vestibular Ocular reflex visual stimulation is not sensitive. In this paper, the ability of visual inhibition of temperature nystagmus in 10 cerebellar disorders was measured and correlated with tracking gain and superimposed glaucoma, and the relationship between vestibular gain and chair speed in addition to eye speed was analyzed. An eye-animal model was then developed to help understand these problem. Results: (1) Temperature nystagmus, 6 cases of 10 cases of inhibition decreased, and some negative. (2) Smooth tracking, 4 cases of 10 cases significantly reduced gain, superimposed glaucoma increased. (3) Vestibular nystagmus, 6 cases of vestibular gain increased but not with the smooth tracking gain.