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永存原始玻璃体增生症(persistent hyperplastic primary vitreous,PHPV)是由于原始玻璃体及玻璃体血管没有消退,继续增殖所导致的玻璃体先天异常。 Reese(1955)首次提出了PHPV这个命名,近年来随着对本病病理学研究的深入,部分学者更倾向于用晶体后纤维血管膜持续增生症(persistent hyperplastic tunica vasculosalentis,PHTVL)来命名局限于前部玻璃体的病变,而仅将伴有后部玻璃体增生的病变称为 PHPV。Goldberg (1997)重新命名PHPV为持续性胎儿血管化(persistent fetal vasculature,PFV),他认为PFV能更好地描述由于胎儿血管未完全退化而导致的诸多临床表现,如晶体后纤维增殖膜、视网膜皱襞、自发性眼底出血等。
Persistent hyperplastic primary vitreous (PHPV) is caused by the original vitreous and vitreous vessels did not subside, continue to proliferate caused by congenital anomalies of the vitreous. Reese (1955) first proposed the PHPV name, with the pathology of the disease in recent years, some scholars prefer to use persistent hyperplastic tunica vasculosalentis (PHTVL) after the name is limited to Anterior vitreous lesions, but only with the rear vitreous hyperplasia lesions called PHPV. Goldberg (1997) renamed PHPV as persistent fetal vasculature (PFV), arguing that PFV can better characterize many of the clinical manifestations that result from incompletely degraded fetal blood vessels such as the posterior lens epithelium, the retina Folds, spontaneous fundus hemorrhage and so on.