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最近看到几篇关于混用溴氰菊酯及防虫磷来防治储粮害虫的报导(张国梁等,1982;檀先昌等,1985;陈嘉东等,1985;),都指出了混用的有效性,二种杀虫剂可以取长补短,例如防虫磷对于谷蠹的毒效不高,而溴氰菊酯对其十分有效,杂拟谷盗及米象对菊酯类杀虫剂都有较高的抵抗力,而防虫磷对其有效,看来,这一混配的剂型是一个比较理想的储粮保护剂。同样的报导在农业害虫及医学昆虫上也有见到,韩喜莱等(北京农业大学1985,见北京农药学会1985年年会报告论文)报道,菜蚜及菜青虫用几种有机磷及几种菊酯类化合物防治时,单用都不如两类杀虫剂的混用,混用有显著的“增效”作用,上海昆虫所对于蚊子的防治,及北京动物所毒理室对于家蝇的室内毒力测定,都证明了有机磷化合物与菊酯类化合物混用时,效果都比单用为好。当时提出混用的根据是:这两类杀虫剂的毒理机制不同。有机磷是乙酰胆碱酯酶的抑制剂(高浓度时对于乙酰胆碱受体也有一些影响),总之是抑制胆碱激性突触的神经传递的。菊酯类杀虫剂的毒理机制虽然还不完全肯定,但可以说它总是一个轴突毒剂,但究竟其作用
Recently, several reports on the use of deltamethrin and pest control to control stored grain pests have been reported (Zhang Guoliang et al., 1982; Tan Xianchang et al., 1985; Chen Jiandong et al., 1985; Insecticides can complement each other’s strengths and weaknesses. For example, pendimethalin is not very effective against Rhizoctonia solani, while deltamethrin is very effective against it. Both trichosanthin and rice elephant have higher resistance to pyrethroid insecticides, Pesticide phosphorus effective for it, it appears that this mixed formulation is an ideal grain protectant. The same report was also found on agricultural pests and medical insects. Han Xilai et al. (Beijing Agricultural University, 1985, see 1985 Annual Conference Report of Beijing Pesticide Society) reported that aphids and cabbage caterpillars were treated with several organic phosphorus and several Control of pyrethroids when compared with the two types of insecticides are not as good as mixed with significant “synergistic” effect, the Shanghai mosquito control of insects, and Beijing animal poisoning room for housefly indoor poisoning Force measurement, all proved organic phosphorus compounds and pyrethrins when mixed, the effect is better than single use. The rationale for the proposed mix was that the toxicological mechanisms of the two classes of pesticides differed. Organophosphorus is an inhibitor of acetylcholinesterase (also has some effects on acetylcholine receptors at high concentrations), in short, it inhibits the neurotransmission of cholinergic synapses. Although the toxicological mechanism of pyrethroid insecticides is not entirely sure, it can be said that it is always an axonal agent, but what exactly does it do?