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三磷酸腺苷(ATP)通过作用于内皮P_2嘌呤受体释放一种内皮衍生的松弛因子(EDRF)而发挥它的血管舒张作用。可能由于其分解迅速,有关活体内ATP作用的研究很少,本文应用半衰期为1h的钴-ATP复合物(Co-ATP)观察了有卒中倾向的自发性高血压兔与健康对照兔的脑血管和代谢变化。
Adenosine triphosphate (ATP) exerts its vasodilatory effect by acting on the endothelial purinergic P2 receptors to release an endothelial-derived relaxing factor (EDRF). It may be due to its rapid decomposition, the role of in vivo ATP little research in this paper, the half-life of 1h Co-ATP complex (Co-ATP) to observe the tendency of stroke spontaneously hypertensive rabbits and healthy control rabbit cerebrovascular And metabolic changes.