通过经口投给100ppm氯化镉与不投给镉的两组Wistar雄性大鼠研究镉对大鼠肠粘膜中三价运铁蛋白形成的影响。结果表明,镉可降低脏器中贮藏铁的含量,可阻止铁进入肠全粘膜中,镉引起动物铁不足与其枯膜中铁蛋白及其它未知蛋白结合的关系应给以重新评价,而在体外试验中,即使给于高浓度氯化镉,对前运铁蛋白转为运铁蛋白也仅仅表现轻微的抑制作用。 The effects of cadmium on the formation of trivalent transferrin in the intestinal mucosa of rats were investigated by oral administration of 100 ppm cadmium chloride and two Wistar rats without cadmium. The results showed that cadmium can reduce the content of stored iron in the organ and prevent iron from entering the intestinal mucosa. The relationship between cadmium-induced iron deficiency and the binding of ferritin and other unknown proteins in the dead membrane should be re-evaluated. In vitro experiments , Only slight inhibition of transferrin to transferrin was observed even with high concentrations of cadmium chloride.