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本文报告了急性缺氧和缺氧习服对冻伤大鼠直肠温度和冻足皮肤温度的影响。结果显示，平原冻伤（ＦＮ）组在冷冻与复温过程中，大鼠直肠温度（Ｔｒ）不变或升高；冻足皮肤温度（Ｔｓ）在冻后１～２天升至高峰，随后降低，冻后６～７天降至接近未冻足水平。在模拟６０００ｍ缺氧条件下，大鼠冷冻前、后的Ｔｒ均降低，但急性缺氧冻伤（ＦＡＨ）组较４ｈ／天连续４周缺氧习服大鼠的缺氧冻伤（ＦＨＡＣ）组降低更明显；复温过程中ＦＡＨ组Ｔｒ继续降低，而ＦＨＡＣ组Ｔｒ不再下降。冻后冻足Ｔｓ变化：ＦＡＨ组与ＦＮ组接近；ＦＨＡＣ组冻后２天内稍升高，随之快速降低，冻后７天仍低于未冻侧。上述结果表明，缺氧使大鼠呈现低体温，缺氧复合一定强度冷刺激使低体温加重；缺氧习服可相对减轻缺氧性低体温的程度，但冻足Ｔｓ减低，外周组织抗冻力减弱。
This article reports the effect of acute hypoxia and hypoxia acclimation on rectal temperature and frozen skin temperature in rats with frostbite. The results showed that the rectal temperature (Tr) was unchanged or increased in the FN group during freezing and rewarming period, and the skin temperature (Ts) peaked at 1 ~ 2 days after freezing, and then decreased , 6 to 7 days after freezing dropped to near the level of non-frozen feet. Trichoderma reesei before and after hypoxia in simulated hypodermic conditions at 6,000 m decreased, but decreased in hypoxic-ischemic injury (FHAC) group compared with 4 h per day for 4 weeks in hypoxic-ischemic rats More obvious; during rewarming FA group Tr continued to decrease, while FHAC group Tr no longer decreased. Frozen foot Ts change after freezing: FAH group and FN group close; FHAC group slightly increased within 2 days after freezing, followed by rapid decline, 7 days after freezing is still lower than the non-frozen side. The results showed that hypoxia rats hypothermia, a certain intensity of hypoxia hypoxia cold hypothermia to aggravate hypoxia; hypoxia habit may be relatively reduced the extent of hypothermia, hypothermia Ts decreased, peripheral tissue antifreeze Weakened power.