对34例体位性窦房、房室传导阻滞进行的临床分析表明:(1)阻滞发生的机制是由于迷走神经功能亢进,引起窦房或房室连接组织的抑制,使其相对不应期延长;而活动性器质性病损所造成的阻滞是由其绝对不应期延长所致。二者是有区别的。(2)阻滞的程度轻,常在Ⅰ°～Ⅱ°Ⅰ型之间;阻滞的部位高,多在窦房或房室连接组织水平,故不影响 Clinical analysis of 34 cases of orthostatic sinus, atrioventricular block showed that: (1) The mechanism of blockage is due to the vagus nerve hyperfunction, causing sinoatrial or atrioventricular tissue inhibition, so that the relative refractory period Prolonged; and active organic disease caused by the block is due to its absolute refractory period due to prolongation. There is a difference between the two. (2) the degree of retardation is light, often between Ⅰ ° ~ Ⅱ ° type Ⅰ; block high, mostly in the sinoatrial or atrioventricular tissue level, it does not affect