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因阴茎组织纤维化引起的勃起功能障碍特点是:血管平滑肌细胞(SMC)凋亡,阴茎海绵体纤维化,海绵体静脉闭塞功能障碍(CVOD)。PDE5抑制剂可通过上调诱生型一氧化氮合酶(NOS2A)来抑制阴茎
Erectile dysfunction due to penile fibrosis is characterized by apoptosis of vascular smooth muscle cells (SMC), cavernous fibrosis, and cavernous venous occlusive dysfunction (CVOD). PDE5 inhibitors inhibit the penis by up-regulating the inducible nitric oxide synthase (NOS2A)