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对180例老年病毒性肝炎检测了血清腺苷脱氨酶(SADA)及单光子发射计算机断层摄影(SPECT),结果表明老年重症肝炎和肝炎后肝硬化(LC)SADA活力显著升高;慢性活动型(CAH)中度升高;急性黄疸型(AH)轻度升高;慢性迁延型(CPH)低度升高,且比非老年病毒性肝炎组有显著性升高(P<0.01或P<0.05)。ECT所测99mTC-EHIDA肝脏主动摄取、分泌、排泄时间和在肝内半衰期,老年重症肝炎和LC显著延长;CAH明显延长;AH轻度延长;CPH略有延长,且较对照组明显延长(P<0.01或P<0.05)。两种检测结果呈正相关,均能准确地反映肝细胞受损的严重程度,并说明老年病毒性肝炎肝功损害尤其严重,预后甚差。
Serum adenosine deaminase (SADA) and single-photon emission computed tomography (SPECT) were detected in 180 elderly patients with viral hepatitis. The results showed that the activity of SADA in elderly patients with severe hepatitis and post-hepatitis cirrhosis (LC) was significantly increased. Chronic activity (CAH) increased moderately; acute jaundice (AH) increased mildly; chronic hypothermia (CPH) increased slightly and was significantly higher than non-elderly viral hepatitis group (P <0.01) Or P <0.05). ECT measured 99mTC-EHIDA liver initiative take, excretion, excretion time and in the liver half-life, severe hepatitis and LC significantly prolonged; CAH was significantly prolonged; mildly prolonged AH; CPH slightly extended, and significantly longer than the control group (P <0.01 or P <0.05). The two test results were positively correlated, can accurately reflect the severity of liver cell damage, and that elderly liver damage is particularly serious viral hepatitis, the prognosis is poor.