Abscisic acid, a promising therapeutic molecule to prevent Alzheimer’s and neurodegenerative disease

来源 :中国神经再生研究(英文版) | 被引量 : 0次 | 上传用户:zxhua2006
下载到本地 , 更方便阅读
声明 : 本文档内容版权归属内容提供方 , 如果您对本文有版权争议 , 可与客服联系进行内容授权或下架
论文部分内容阅读
Neuroinflammatory processes induce neuronal damage and underlie the onset of neurodegenerative pathologies, including Alzheimer’s disease (AD) (Calsolaro and Edison, 2016). Sev-eral conditions, ranging from emotional stress to metabolic syndrome, including traumatic brain injury, chronic infections or gut microbiota disruption can cause or aggravate neuroin-flammation. Moreover, aging is the major factor associated with neurodegenerative diseases.
其他文献
Alpinia oxyphylla, a traditional herb, is widely used for its neuroprotective, antioxidant and memory-improving effects. However, the neu-roprotective mechanism
Autophagy has been shown to have a protective effect against brain damage. Ligustilide (LIG) is a bioactive substance isolated from Ligu-sticum chuanxiong, a tr
The locus coeruleus is a pontine nucleus that produces much of the brain’s norepinephrine. Despite its small size, the locus coeruleus is critical for a myriad
The details of adult neurogenesis, including environmental triggers, region specificity, and species ho-mology remain an area of intense investigation. Slowing
Mitochondrial dysfunction in neurons has been implicated in hypoxia-ischemia-induced brain injury. Although mesenchymal stem cell ther-apy has emerged as a nove
Understanding the mechanisms underlying cell-surface interaction is of fundamental importance for the rational design of scaffolds aiming at tissue engineering,
Over the last ten years or so, it has become apparent that pericytes have the potential to differentiate into other cell types which may help in the repair and
期刊
Repetitive magnetic stimulation is effective for treating posttraumatic neuropathies following spinal or axonal injury. Neurotropin is a potential treatment for
At present, there is no reliable biomarker for the diagnosis of traumatic brain injury (TBI). Studies have shown that extracellular vesicles released by damaged
The contribution of an impaired astrocytic K+ regulation system to epileptic neuronal hyperexcitability has been increasingly recognized in the last decade. A d