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一般诊断急性穿壁性心肌梗塞并不困难。当临床心电图上发现有连续 ST 段抬高,继之出现病理性 Q 波和 T 波倒置,再加上有胸痛病史,便足可以确诊。酶类方面,肌酸磷酸激酶(CPK),血清谷草转氨酶(SGOT)及乳酸脱氢酶(LDH)的增高,与其说对检出穿壁性心肌梗塞的重要,不如说对判断穿壁性心肌梗塞的阶段(超急性期、急性期、亚急性期),或许对判断范围更为重要。酶曲线是心肌坏死的病理特征,但是,既使没有酶曲线,连续心电图改变也可诊断穿壁性心肌梗塞,而它发生的时间则可根据临床发作时间估计。
General diagnosis of acute transmural myocardial infarction is not difficult. When the clinical ECG found on a continuous ST segment elevation, followed by pathological Q wave and T wave inversion, coupled with a history of chest pain, foot enough to confirm. Enzymes, creatine phosphokinase (CPK), serum aspartate aminotransferase (SGOT) and lactate dehydrogenase (LDH) increase, not so much for the detection of transmural myocardial infarction important, as it is to judge the transmural myocardium The stage of infarction (hyperacute, acute, and subacute phases) may be more important in determining the extent of the disorder. The enzyme profile is a pathological feature of myocardial necrosis, but even without an enzyme profile, continuous ECG changes can be diagnosed with transmural myocardial infarction, and its timing can be estimated based on clinical onset time.