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目的:提出上睑下垂病因学的探讨。方法:本文观察7例上睑下垂患者的提上睑肌的超微病理改变。结果:电镜显示提上睑肌纤维呈不同程度的发育异常、变性、坏死,粗丝、细丝失去其正常排列;Muller平滑肌虽然无明显变性,但共超微结构显示发育不良,兼有平滑肌与骨骼肌两者的结构特点。结论:上睑下垂的病因除提上睑肌没有发育成熟或发育异常,Muller平滑肌亦有发育异常而失去正常功能。并且讨论了上睑下垂的分类。
Objective: To investigate the etiology of ptosis. Methods: This paper was to observe the ultrastructural changes of levator muscle in 7 patients with ptosis. Results: Electron microscopy showed that the levator muscle fibers developed different degrees of dysplasia, degeneration and necrosis, thick filaments and filaments lost their normal alignment. Although the Muller smooth muscle showed no obvious degeneration, the total ultrastructure showed dysplasia with both smooth muscle and skeletal Muscle structure characteristics of both. CONCLUSION: The etiology of ptosis, in addition to the absence of developmental maturity or dysplasia of the levator palpebra, the abnormal development of the Muller smooth muscle and loss of normal function. And discussed the classification of ptosis.