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Purpose: To investigate whether the over-expression of PRMT6 can attenuate the development of inflammation, apoptosis, oxidative stress in CSE-induced emphysema in a mice model.Methods: 32 BALB/c mice were randomly divided into 4 groups.We measured lung function and pathology of lung tissue.The expression of protein was detected in lung homogenates by Western-blotting.The mRNA expression was measured by RT-PCR.