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  5. eNOS modified endothelial progenitor cells inhibit efficiently neointima formation and enhancement o

eNOS modified endothelial progenitor cells inhibit efficiently neointima formation and enhancement o

来源 :第十二届中国介入心脏病学大会(CIT2014) | 被引量 : 0次 | 上传用户：bosimao_wang
【摘 要】
：
Background Loss of endothelial NO production after arterial injury may contribute to restenosis,characterized by neointima formation and elastic recoil.Previous studies have established that bone marr
【作 者】
：
Cui Bin Huang Lan Jin Jun Song Minbao Chen Jianfei 
【机 构】
：
Institute of Cardiovascular Disease of PLA,Xin Qiao Hospital,Third Military Medical University,Chong
【出 处】
：
第十二届中国介入心脏病学大会(CIT2014)
【发表日期】
：
2014年3期
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Background Loss of endothelial NO production after arterial injury may contribute to restenosis,characterized by neointima formation and elastic recoil.Previous studies have established that bone marrow-derived endothelial progenitor cells (EPCs) play an important role in vascular repair.In this study,we investigated that hypothesis that overexpression of vasculoprotective gene endothelial nitric oxide synthase (eNOS) in EPCs may restore NO production and inhibit neointimal hyperplasia.
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