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关于补体系统在胰岛素依赖型糖尿病(IDDM)发病机制中的作用,有4种观点:①补体直接参与胰岛β细胞的溶解破环;②有微血管病时补体才出现溶解β细胞的作用;③补体系统清除免疫复合物的功能障碍,免疫复合物沉积是微血管病的部分原
There are four perspectives on the role of the complement system in the pathogenesis of insulin-dependent diabetes mellitus (IDDM): (1) complement is directly involved in the islet β-cell disruption and disruption; (2) there is a role for the complement to dissolve the β-cell in the presence of microangiopathy; The system removes dysfunction of immune complexes, deposition of immune complexes is part of the original microangiopathy