论文部分内容阅读
[目的]研究外源SA对高温强光胁迫下小麦叶片类囊体膜D1蛋白磷酸化和光合机构运转的影响。[方法]用0.5 mmol/L水杨酸(SA)溶液预处理灌浆期小麦叶片,以水预处理为对照,将预处理植株进行高温强光[35℃,1600μmol/(m2.s)]胁迫,测定胁迫处理过程中小麦旗叶活性氧代谢、叶绿素荧光参数及D1蛋白的变化。[结果]SA预处理有效抑制了高温强光下D1蛋白的净降解,保持了较高的D1蛋白磷酸化水平,高效防护了高温强光所致的氧化损伤,维持较高的超氧化物岐化酶(SOD)和抗坏血酸过氧化物酶(APX)活性,维持了较高的通过PSⅡ电子传递速率(Fv/Fo)、PSⅡ原初光化学效率(Fv/Fm)、实际光化学效率(ФPSⅡ)、光化学猝灭系数(qP)。[结论]SA预处理明显减轻了高温强光胁迫对光合机构的损伤,保证了PSⅡ的正常运转。
[Objective] The research aimed to study the effects of exogenous SA on the phosphorylation of D1 protein and the photosynthetic machinery in thylakoid membrane of wheat under high temperature and high light stress. [Method] With 0.5 mmol / L salicylic acid (SA) solution pretreatment of wheat leaves at grain filling stage, water pretreatment as a control, the pretreated plants under high temperature and high light [35 ℃, 1600μmol / (m2.s)] , And the changes of active oxygen metabolism, chlorophyll fluorescence parameters and D1 protein in wheat flag leaf during stress treatment were measured. [Result] SA pretreatment effectively inhibited the net degradation of D1 protein under high temperature and high light, maintained the high level of D1 protein phosphorylation, effectively protected the oxidative damage induced by high temperature and high light, and maintained high superoxide dismutase (SOD) and ascorbate peroxidase (APX), and maintained high Fv / Fo, PSv primary photochemical efficiency (Fv / Fm), actual photochemical efficiency (ФPSⅡ), photochemical Quenching coefficient (qP). [Conclusion] SA pretreatment significantly reduced the damage of photosynthetic apparatus under high temperature and high light stress and ensured the normal operation of PSII.