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对１８条缺血性心肌模型犬中发生室性心动过速（简称室速）的６条犬采用组合双极电极记录心肌局部电图，进一步探讨缺血性心脏病室速的机制。结果显示：①缺血区各层心肌电图和心外膜局部电图上均出现延迟电位及碎裂波。②室速的激动顺序为缺血区心内膜（作参照，为０ｍｓ）、缺血区心外膜（１０±０．１０ｍｓ）、边缘区心内膜（１０±０．１２ｍｓ）、边缘区心外膜（１６±０．２０ｍｓ）、正常区心内膜（２０±０．５０ｍｓ）、正常区心外膜为（２７±０．２０ｍｓ）。③当体表ＩＩ导联心电图和心外膜、心肌局部电图均出现心室颤动时，心内膜仍表现为室速的图形。认为室速是多平面多折返所形成的“立体折返”激动的结果；如何寻找折返环入口作为射频消融治疗的靶点至关重要
Six dogs with ventricular tachycardia (VT) in 18 ischemic myocardium models were used to record myocardial electrocardiogram (ECG) by combining bipolar electrodes to explore the mechanism of ventricular tachycardia in ischemic heart disease. The results showed that: (1) delayed potentials and fragmentation wave appeared on the myocardial electrocardiogram and epicardial electrograms in all layers of ischemic area. (2) The ventricular tachyarrhythmia was induced by ischemic endocardium (referenced to 0 ms), ischemic epicardium (10 ± 0.10 ms), marginal zone endocardium (10 ± 0.12 ms), marginal zone The epicardium (16 ± 0.20 ms), normal endocardium (20 ± 0.50 ms) and normal epicardium (27 ± 0.20 ms). ③ When the body surface lead II ECG and epicardial, myocardial electrocardiogram showed ventricular fibrillation, the ventricular endocardial still showed a graph of VT. It is considered that the VT is the result of “three-dimensional reentry” excited by multiplanar reentry. How to find the reentrant ring entrance is the target of radiofrequency ablation