Aβ-Stimulated BMDCs combined with spleen-derived cells attenuates learning and memory deficits and p

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Anti-Aβ immunotherapy could reduce amyloid plaques and amyloid-associated pathologies, attenuate learning and memory deficits in AD animal models.Immune senescence has been proved to play a crucial role in AD pathogenesis,which affects anti-A3 immunotherapv possibly.
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